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Difference between revisions of "Trigeminal Nociceptive Evaluation in TMD Patients by studying CO2-Laser Evoked Potentials and Masseter Laser Silent Periods"
Trigeminal Nociceptive Evaluation in TMD Patients by studying CO2-Laser Evoked Potentials and Masseter Laser Silent Periods (view source)
Revision as of 12:17, 14 October 2024
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===Introduction=== | ===Introduction=== | ||
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In conclusion, this study demonstrated that in TMD patients with unilateral chronic pain, both nociceptive inputs directed to the cortex and the brainstem nociceptive reflex circuit are inhibited. While these results suggest a dysfunction of the nociceptive system that mediates and integrates phasic nociceptive inputs, it is not possible to assert whether this dysfunction plays a role in the pathophysiology of TMD or if it is rather a consequence of chronic pain. | In conclusion, this study demonstrated that in TMD patients with unilateral chronic pain, both nociceptive inputs directed to the cortex and the brainstem nociceptive reflex circuit are inhibited. While these results suggest a dysfunction of the nociceptive system that mediates and integrates phasic nociceptive inputs, it is not possible to assert whether this dysfunction plays a role in the pathophysiology of TMD or if it is rather a consequence of chronic pain. | ||
{{Bib}} | {{Bib}} | ||